MethylFolate 1000 MCG, #60

$ 25.00

Methylmax Substitution. Methylfolate is the actual B9 Vitamin, not Folate or Folic Acid (the drug "vitamin.") Over 50% of the population of the world has...

SKU: MTF
Description

Methylmax Substitution.

Methylfolate is the actual B9 Vitamin, not Folate or Folic Acid (the drug "vitamin.") Over 50% of the population of the world has this MTHFR deficiency which causes the intake of Folate or Folic Acid to become toxic to their body by causing an increased inability to metabolize Homocysteine. The toxicity of Folate and/or Folic Acid is thus caused by the inability of those individuals who have a Genetic SNP ("snip") MTHFR 677 and/or 1298 Homozygous and Heterozygous state of the enzyme deficiency to convert the drug "vitamin" to the actual Metlylfolate Vitamin, the actual B9 Vitamin. This genetic SNP would be meaningless if the NIH March of Dimes group and/or Medical doctors and nutritionists understood biochemistry well enough to stop the harmful behavior of putting the drug vitamin in "fortified" foods and most B vitamins sold in the world today.

Unfortunately, most people and doctors are unaware of this SNP prevalence and even more unaware of how to treat and the consequences of being continuously exposed to the drug "vitamin" called B9. Over 30 specific diseases are increased by this issue including over 80% of all those individuals with emotional or mental disorders which are largely correctable by making the correct diagnosis and by providing the correct vitamin B9. 

Why is this important specifically, the elevation of Homocysteine above a level of 8.0 (well below the laboratory normal range) is associated with an increase in inflammation throughout the body and specifically the elevation of Homocysteine which is associated with an increase in ADMA (Asymmetrical Dimethlyarginine). The elevation of ADMA is known to be associated with ASCVD, i.e., "hardening of the arterial walls" and loss of vascular elasticity thus increasing heart disease. This issue is thus a major cause of disease, i.e., being caused by the drug "vitamin" B9.

Therefore, one may conclude that due to the ignorance of most physicians, NIH, and nutritionists about this issue is contributing to a significant amount of disease simply caused by taking or being exposed to the drug "vitamin" B9 which does not exist naturally. Natural sources of B9 are green leafy vegetables containing the actual vitamin B9, i.e., Methlyfolate. 

If one is a carrier (heterozygous) or homozygous for this condition, simply taking the correct vitamin B9 for life or eating green leafy vegetables and avoiding Folate or Folic Acid (the drugs, not naturally occurring) may help prevent the increase in Homocysteine and thus ADMA and thus prevent the development of numerous physical and mental ailments and chronic diseases related to the elevation of Homocysteine, neurotransmitter imbalances, and increases in vascular disease related to ADMA.

How simple is that? It is very sad that more physicians, especially those involved in the treatment of mental health issues remain unaware of this condition.    

These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.